Search results for " muscle weakness"

showing 8 items of 8 documents

Dermatomyositis induced by the secretion of Phyllomedusa bicolor or Kambô frog - A case report

2020

The Amazonian Kambô frog, Phyllomedusa bicolor, is commonly known for the potential benefits of its secretion. The Kambô ritual consists in applying the toxin directly to a freshly burnt skin area, since it is believed by natives and shamans to purify and strengthen the body and mind of the user. We describe a 33-year old female with a history of periodic use of Kambô toxin who presented with a 3-week course of asthenia, malaise, myalgia, and proximal muscle weakness predominantly in the lower limbs. She had elevated muscle enzymes and an abnormal electromyography. We used the 2017 European League Against Rheumatism and American College of Rheumatology classification criteria to establish t…

Adult0106 biological sciencesmyalgiamedicine.medical_specialtyProximal muscle weaknessRanidaePhyllomedusa bicolorToxicology01 natural sciencesDermatomyositisMalaise03 medical and health sciencesPrednisoneInternal medicineAnimalsHumansMedicine0303 health sciencesbiologybusiness.industry010604 marine biology & hydrobiology030302 biochemistry & molecular biologyDermatomyositismedicine.diseasebiology.organism_classificationDermatologyRheumatologyFemalemedicine.symptombusinessRheumatismmedicine.drugToxicon
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Lower limb muscle weakness predicts use of a multiple- versus single-step strategy to recover from forward loss of balance in older adults.

2012

BACKGROUND: Older adults compared with young adults have reduced strength and balance recovery ability. The purpose of the present study was to investigate whether age, sex, and/or lower limb strength predicted the stepping strategy used to recover from a forward loss of balance. METHODS: Ninety-five, community-dwelling, older adults, aged 65-90 years, participated in the study. Loss of balance was induced by releasing participants from a static forward lean. Participants performed four trials at three initial lean magnitudes and were subsequently classified as using a single- or multiple-step strategy. Isometric strength of the ankle, knee, and hip joint flexors and extensors was assessed …

AdultMalemedicine.medical_specialtyAgingMovementPoison controlIsometric exerciseLogistic regressionRisk AssessmentPhysical medicine and rehabilitationSex FactorsPredictive Value of TestsmedicineOdds RatioReaction TimeHumansProspective StudiesMuscle SkeletalPostural BalanceBalance (ability)AgedAged 80 and overAnalysis of VarianceMuscle Weaknessbusiness.industryLower limb muscle weaknessAge FactorsMuscle weaknessStepwise regressionmedicine.anatomical_structureLogistic ModelsLower ExtremitySensation DisordersPhysical therapyAccidental FallsFemaleGeriatrics and Gerontologymedicine.symptomAnklebusinessThe journals of gerontology. Series A, Biological sciences and medical sciences
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Intermediate Filament Diseases: Desminopathy

2008

Desminopathy is one of the most common intermediate filament human disorders associated with mutations in closely interacting proteins, desmin and alphaB-crystallin. The inheritance pattern in familial desminopathy is characterized as autosomal dominant or autosomal recessive, but many cases have no family history. At least some and likely most sporadic desminopathy cases are associated with de novo DES mutations. The age of disease onset and rate of progression may vary depending on the type of inheritance and location of the causative mutation. Typically, the illness presents with lower and later upper limb muscle weakness slowly spreading to involve truncal, neck-flexor, facial and bulba…

GeneticsPathologymedicine.medical_specialtyPoint mutationMutantCardiomyopathyIntermediate Filamentsalpha-Crystallin B ChainGene mutationBiologymedicine.diseaseSudden deathPolymorphism Single NucleotideArticleUpper limb muscle weaknessDesminMuscular DiseasesmedicineDisease ProgressionAnimalsHumansDesminIntermediate filament
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Early-infantile onset epilepsy and developmental delay caused by bi-allelic GAD1 variants

2020

Mice lacking GAD1 show neonatal mortality, but the human phenotype associated with GAD1 disruption is poorly characterized. Neuray et al. describe six patients with biallelic GAD1 mutations, presenting with early-infantile onset epilepsy, neurodevelopmental delay, muscle weakness and non-CNS manifestations.

Male0301 basic medicineGlutamate decarboxylaseMalalties cerebralsNeurotransmissorsNeurodevelopmental delayEpilepsy0302 clinical medicineMESH: ChildAge of OnsetChildcleft palateGAD1AcademicSubjects/SCI01870Glutamate DecarboxylaseGlutamate receptorMuscle weakness//purl.org/becyt/ford/3.1 [https]NeurotransmittersMESH: InfantHypotoniamuscle weakneCleft palateMESH: EpilepsyChild PreschoolMuscle Hypotonia[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]//purl.org/becyt/ford/3 [https]FemaleBrain diseasesAbnormalitiesmedicine.symptomMultiplemedicine.drugcleft palate; epilepsy; GAD1; muscle weakness; neurodevelopmental delayMESH: Glutamate Decarboxylasemedicine.medical_specialtyMESH: Abnormalities MultipleMESH: MutationMESH: Age of OnsetBiologyInhibitory postsynaptic potentialGAD1 cleft palate epilepsy muscle weakness neurodevelopmental delay.gamma-Aminobutyric acidGAD1neurodevelopmental delay03 medical and health sciencesExcitatory synapseInternal medicinemedicineHumansAbnormalities MultiplePreschoolAllelesMESH: Neurodevelopmental Disordersmuscle weaknessMESH: HumansEpilepsyMESH: Muscle HypotoniaMESH: AllelesMESH: Child PreschoolInfantmedicine.diseaseMESH: MaleEpilèpsiaEditor's Choice030104 developmental biologyEndocrinologyNeurodevelopmental DisordersMutationepilepsyAcademicSubjects/MED00310Neurology (clinical)Cleft palate; Epilepsy; GAD1; Muscle weakness; Neurodevelopmental delay; Abnormalities Multiple; Age of Onset; Alleles; Child; Child Preschool; Epilepsy; Female; Glutamate Decarboxylase; Humans; Infant; Male; Muscle Hypotonia; Mutation; Neurodevelopmental DisordersMESH: Female[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology030217 neurology & neurosurgeryReports
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Muscular performances at the ankle joint in young and elderly men.

2005

The effect of aging on mechanical and electromyographic characteristics of ankle joint muscles was investigated in 11 young (mean age 24 years) and 12 elderly (mean age 77 years) males. Maximal and submaximal isometric voluntary torques were measured during ankle plantarflexion and dorsiflexion. Electromyographic activities of triceps surae and tibialis anterior muscles were recorded. The elderly group developed equal maximal dorsiflexion torques (42 vs 45 N.m, p >.05), but in plantarflexion, the elderly group was weaker (80 vs 132 N.m, p <.001) and presented a decreased twitch amplitude (11 vs 16 N.m) and lower coactivation (8% vs 15%) than that of the young adults. We established a linear…

MaleAgingMESH: Range of Motion ArticularMESH : Ankle Joint[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH : Electric StimulationMESH: Muscle ContractionMESH : Muscle WeaknessMESH : AgedMESH: Evoked Potentials MotorIsometric exerciseElectromyographyMESH : Evoked Potentials Motor0302 clinical medicineMESH: Ankle JointMESH: AgingMESH: Isometric ContractionRange of Motion ArticularYoung adultMESH : Muscle Skeletal10. No inequalityMESH: AgedMESH: Muscle SkeletalMuscle Weaknessmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Muscle WeaknessMESH: Electric StimulationMESH : AdultCoactivationMESH: Legmedicine.anatomical_structureLinear relationshipMESH : ElectromyographyRange of motionMuscle ContractionAdultmedicine.medical_specialtyMESH : MaleMESH: Research Support Non-U.S. GovMESH: ElectromyographyMESH : Isometric Contraction03 medical and health sciencesPhysical medicine and rehabilitationIsometric Contractionmedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansMESH : Research Support Non-U.S. GovMuscle SkeletalAgedLegMESH: HumansElectromyographybusiness.industryMESH : HumansMean ageMESH: Adult030229 sport sciencesEvoked Potentials MotorElectric StimulationMESH : AgingMESH: MaleMESH : Range of Motion ArticularPhysical therapyMESH : Muscle ContractionTibial NerveGeriatrics and GerontologyAnkleMESH : Legbusinesshuman activitiesAnkle Joint030217 neurology & neurosurgery
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HACD1, a regulator of membrane composition and fluidity, promotes myoblast fusion and skeletal muscle growth

2015

International audience; The reduced diameter of skeletal myofibres is a hallmark of several congenital myopathies, yet the underlying cellular and molecular mechanisms remain elusive. In this study, we investigate the role of HACD1/PTPLA, which is involved in the elongation of the very long chain fatty acids, in muscle fibre formation. In humans and dogs, HACD1 deficiency leads to a congenital myopathy with fibre size disproportion associated with a generalized muscle weakness. Through analysis of HACD1-deficient Labradors, Hacd1-knockout mice, and Hacd1-deficient myoblasts, we provide evidence that HACD1 promotes myoblast fusion during muscle development and regeneration. We further demons…

Male[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process EngineeringCellular differentiationGeneralized muscle weaknessBiologyMuscle Developmentcentronuclear myopathyCell LineMyoblasts03 medical and health scienceschemistry.chemical_compoundMyoblast fusionMice0302 clinical medicineDogsVLCFA[SDV.IDA]Life Sciences [q-bio]/Food engineeringGeneticsmedicineMyocyteAnimalsHumans[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringMUFACentronuclear myopathyMuscle SkeletalMolecular Biology030304 developmental biologyMice Knockout0303 health sciencesPTPLACell MembraneSkeletal muscleCell DifferentiationCell BiologyGeneral MedicineArticles[SDV.IDA] Life Sciences [q-bio]/Food engineeringmedicine.diseaseCongenital myopathyLysophosphatidylcholinemedicine.anatomical_structureLPCchemistryBiochemistryFemaleProtein Tyrosine Phosphatasescentronuclear myopathy;lpc;mufa;ptpla;vlcfa030217 neurology & neurosurgery
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Assessment of quadriceps muscle weakness in patients after total knee arthroplasty and total hip arthroplasty: methodological issues.

2013

The aim of this exploratory study was to verify whether the evaluation of quadriceps muscle weakness is influenced by the testing modality (isometric vs. isokinetic vs. isoinertial) and by the calculation method (within-subject vs. between-subject comparisons) in patients 4-8months after total knee arthroplasty (TKA, n=29) and total hip arthroplasty (THA, n=30), and in healthy controls (n=19). Maximal quadriceps strength was evaluated as (1) the maximal voluntary contraction (MVC) torque during an isometric contraction, (2) the peak torque during an isokinetic contraction, and (3) the one repetition maximum (1-RM) load during an isoinertial contraction. Muscle weakness was calculated as the…

Malemedicine.medical_specialtyWeaknessJoint replacementmedicine.medical_treatmentArthroplasty Replacement HipBiophysicsNeuroscience (miscellaneous)Total knee arthroplastyIsometric exerciseMuscle Strength DynamometerQuadriceps MusclePhysical medicine and rehabilitationOne-repetition maximumIsometric ContractionmedicineHumansIn patientArthroplasty Replacement KneeMuscle SkeletalAgedMuscle Weaknessbusiness.industryQuadriceps muscle weaknessMuscle weaknessReproducibility of ResultsMiddle AgedTorquePhysical therapyFemaleNeurology (clinical)medicine.symptombusinessJournal of electromyography and kinesiology : official journal of the International Society of Electrophysiological Kinesiology
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Clinical and Genetic Aspects of Juvenile Onset Pompe Disease

2021

AbstractLittle is known about clinical symptomatology and genetics of juvenile onset Pompe disease (JOPD). The aims of this study were to analyze how these children are diagnosed, what clinical problems they have, and how phenotype is related to genotype. To accomplish this, we analyzed retrospectively data of 34 patients diagnosed after their first and before completion of their 18th birthday. Median age at diagnosis was 3.9 (range 1.1–17) years. Eight patients (23.5%) developed initial symptoms in the first year, 12 (35%) between 1 and 7 years, and 6 (18%) thereafter. Eight (23.5%) had no clinical symptoms at the time of diagnosis. Indications for diagnostics were a positive family histor…

Pediatricsmedicine.medical_specialtyGeneralized muscle weaknessDisease03 medical and health sciences0302 clinical medicineGenotypeHumansMedicineFamily historyRetrospective Studies030304 developmental biology0303 health sciencesGlycogen Storage Disease Type IIbusiness.industryHypertrophic cardiomyopathyMuscle weaknessalpha-GlucosidasesGeneral Medicinemedicine.disease3. Good healthPhenotypeJuvenile onsetMutationPediatrics Perinatology and Child HealthFailure to thriveNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgeryNeuropediatrics
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